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We investigated the role of eicosanoids in the regulation of serum progesterone in ovariectomized rats. After ovariectomy, serum progesterone levels decreased in rats with or without estradiol replacement and were restored by the treatment with the specific cyclooxygenase (COX) inhibitors, indomethacin (IND) or nimesulide. The COX inhibitors were also shown to alter the percentage of animals which displayed corpus luteum (CL) regression and reduce serum progesterone levels in ovariectomized rats primed with estradiol. These results indicate that COX-derived products are involved in the regulation of preovulatory progesterone. In contrast, treatment with the 5-lipoxygenase inhibitor, zileuton, reduced serum progesterone levels in ovariectomized rats without estradiol treatment, but had no effect on CL regression. To examine the role of endogenous prostaglandins on preovulatory progesterone production in vivo, rats were treated with the prostaglandin synthesis inhibitor, flurbiprofen (10 mg/kg/day, s.c., days -2 to 0). Flurbiprofen completely inhibited endogenous prostaglandin biosynthesis for at least 96 h after onset of treatment. Inhibition of COX activity did not affect the percentage of animals which displayed CL regression or serum progesterone levels, while the percentage of animals which displayed CL regression was reduced by the inhibition of 5-lipoxygenase activity. These data indicate that prostaglandin synthesis induced by endogenous progesterone does not contribute to the regulation of preovulatory progesterone levels in ovariectomized rats.Update
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