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Despite widespread access to combination antiretroviral therapy (cART), HIV infection continues to result in significant morbidity and mortality. Optimal treatment strategies have been defined to control viral replication to undetectable levels and to preserve immune function. However, the immune system of patients with HIV-1 infection is often compromised, and patients who are on cART may exhibit elevated levels of immunological markers of inflammation, immune activation and immune senescence. These markers are associated with CD4 T-cell depletion, elevated risk of cardiovascular and non-AIDS-related mortality and may predict clinical outcome in patients with HIV-1 infection. Further, it is possible that persistent immune activation may promote the development of certain AIDS-related complications, including tuberculosis and other opportunistic infections. The potential to modify and control chronic inflammation in patients with HIV-1 infection by interrupting virus replication is being actively explored. In addition to these mechanistic studies, clinical trials are in progress to determine whether cART initiated during chronic immune activation is associated with increased incidence of comorbid conditions. It is increasingly apparent that the period of disease progression in HIV-1 infection coincides with the time at
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