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AMQ is a potent and selective inhibitor of monoamine oxidase-A (MAO-A). The present study investigated the in vivo metabolism of [carbonyl-11C]AMQ in rat brain. At 5 min after an intravenous administration of [carbonyl-11C]AMQ to rats (n = 2), radioactivity was detected in the cerebral cortex, hypothalamus, striatum, thalamus, hippocampus and cerebellum. The radioactivity in these structures was gradually cleared, and reached a plateau level at 60 min. [11C]AMQ and two of its metabolites were identified in the rat brain using HPLC and radio-HPLC. The metabolites were identified by comparison of their chromatographic retention times and their radioactivities with those of synthetic [11C]AMQ and its reference products. Among them, [11C]AMQ-4-acetic acid was found to be the predominant metabolite. On the basis of these data, a HPLC/radio-HPLC assay was developed to measure the concentration of [11C]AMQ-4-acetic acid in the rat brain. [11C]AMQ-4-acetic acid represented 31 +/- 4% (mean +/- SE) of the administered dose of [11C]AMQ in rat brain at 60 min after the injection.The Aquarian
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